Poster Presentation 1B-36

 

The Effect of Glycosylation on the Thermostability and Activity of Trichoderma reesei Cel7A (CBH I)

 

Tina Jeoh,* William S. Adney, Yat-Chen Chou, David Templeton,

John O. Baker, and Michael E. Himmel

 

National Bioenergy Center

National Renewable Energy Laboratory

1617 Cole Boulevard

Golden, CO 80401

Phone:  (303)384-7777

Fax:  (303)384-7752

E-mail:  tina_jeoh@nrel.gov

 

 

Trichoderma reesei Cel7A (CBH I) is generally considered to be one of the most important industrial cellulase component enzymes due to its efficiency at hydrolyzing insoluble cellulose.  In its native form, Cel7A is a glycoprotein, consisting of four putative N-glycosylation sites at positions 45, 64, 270 and 384 on the catalytic domain

.  Only three of the four sites (45, 270 and 384), however, have been found to be glycosylated.  It has been shown that the extent of glycosylation of Cel7A can vary when expressed in heterologous host cells, such as insect cells or other fungi.  For example, recombinant T. reesei Cel7A expressed in Aspergillus awamori was determined to have five times more glycan content than native T. reesei Cel7A (5.47 ± 0.16 % and 0.84 ± 0.17 % glycan for A. awamori expressed rCel7A and native T. reesei Cel7A, respectively).  Binding and hydrolysis studies using fluorescence-labeled Cel7A and rCel7A, however, showed no activity differences between the two cellulase forms when acting on bacterial cellulose.  Glycosylation is known to affect the secretion, thermal stability, and activity of enzymes, but little is known about the effect of glycosylation on Cel7A.  We present a preliminary study of the effect of glycosylation on the thermal stability and cellulose hydrolysis kinetics displayed by native and various engineered forms of T. reesei Cel7A, including three mutants: N45A, N270A, and N384A.  Tools used to compare these enzyme forms include differential scanning microcalorimetry for overall thermal tolerance, titration microcalorimetry for binding enthalpies, and hydrolysis kinetics on bacterial cellulose.